HRSB Resources and Publications
HRSB Biennial Reports
Utilization of Breast Cancer Research and Education Funds Reports
Related Links[19]
- New York State Department of Health: Funding Opportunities[20]
- New York State Department of Environmental Conservation, Pesticide Reporting Law[21]
- Pesticide Sales and Use Reporting (PSUR) Database[22]
- New York State Grants Reform[23]
- New York State Statewide Financial System[24]
- National Institutes of Health[25]
- National Institutes of Health: Clinical Trials[26]
- PubMed[27]
External Publications
Since 1996, the HRSB’s mission has been to support innovative breast cancer scientific research and education projects within New York State. HRSB has recommended approximately $27 million in research awards through 2025 to some of New York State's finest research teams. Research listed in the table below is a result of this funding.
| Organization | Investigator(s) | Link/PubMed ID# | Publication/Abstract Title |
| Stony Brook University | Chung, Jun | 38201253[28] | The Sensitization of Triple-Negative Breast Cancers to Poly ADP Ribose Polymerase Inhibition Independent of BRCA1/2 Mutation Status by Chemicall Modified microRNA-489 |
| Stony Brook University | Chung, Jun | 39201393[29] | Recent Technologies towards Diagnostic and Therapeutic Applications of Circulating Nucleic Acids in Colorectal Cancers |
| Albany Medical College | Barroso, Margarida | 39642392[30] | Modeling intratumor heterogeneity in breast cancer |
| Albany Medical College | Barroso, Margarida | 13484512[31] | Iron–endoplasmic reticulum–extracellular matrix axis regulates cancer cell invasion |
| Icahn School of Medicine at Mount Sinai | Irie, Hanna | 39541980[32] | A PROTAC degrader suppresses oncogenic functions of PTK6 inducing apoptosis of breast cancer cells |
| Icahn School of Medicine at Mount Sinai | Silva, Jose | 34633138[33] | miR-424/503 modulates Wnt/β-catenin signaling in the mammary epithelium by targeting LRP6 |
| New York University | David, Gregory | bioRxiv[34] | The chromatin associated Sin3B is a critical regulator of DNA damage repair through the engagement of the Non-Homologous End Joining pathway |
| New York University | David, Gregory | bioRxiv[35] | Lipocalin 2 upregulation mediates the detrimental effects of therapy-induced senescence in breast cancer cells |
| New York University | David, Gregory | 35500379[36] | The origins of cancer cell dormancy |
| New York University | David, Gregory | 37314748[37] | Chromatin-associated SIN3B protects cancer cells from genotoxic stress-induced apoptosis and dictates DNA damage repair pathway choice |
| New York University | David, Gregory | 35953598[38] | Therapy-induced senescence promotes breast cancer cells plasticity by inducing Lipocalin-2 expression. |
| Stony Brook University | Chung, Jun | 38201253[39] | The Sensitization of Triple-Negative Breast Cancers to Poly ADP Ribose Polymerase Inhibition Independent of BRCA1/2 Mutation Status by Chemically Modified microRNA-489 |
| Stony Brook University | Chung, Jun | 37892162[40] | Combination Treatment Strategies to Overcome PARP Inhibitor Resistance |
| Stony Brook University | Chung, Jun | 39201393[41] | Recent Technologies towards Diagnostic and Therapeutic Applications of Circulating Nucleic Acids in Colorectal Cancers |
| Albany Medical College | Barroso, Margarida | 39642392[42] | Modeling intratumor heterogeneity in breast cancer |